Frank-Bertoncelj, M. et al. Epigenetically-driven anatomical diversity of synovial fibroblasts guides joint-specific fibroblast functions. Nat. Commun. 8, 14852 (2017).
Google Scholar
Ciurea, A. et al. Joint-level responses to tofacitinib and methotrexate: a post hoc analysis of data from ORAL Start. Arthritis Res. Ther. 25, 185 (2023).
Google Scholar
Elhai, M. et al. The long non-coding RNA HOTAIR contributes to joint-specific gene expression in rheumatoid arthritis. Nat. Commun. 14, 8172 (2023).
Google Scholar
Zhang, F. et al. Deconstruction of rheumatoid arthritis synovium defines inflammatory subtypes. Nature 623, 616–624 (2023).
Google Scholar
Whyte, J. L., Smith, A. A. & Helms, J. A. Wnt Signaling and Injury Repair. Cold Spring Harb. Perspect. Biol. 4, a008078 (2012).
Google Scholar
Chang, M. H. et al. Arthritis flares mediated by tissue-resident memory T cells in the joint. Cell Rep. 37, 109902 (2021).
Google Scholar
Heckert, S. L. et al. Patterns of clinical joint inflammation in juvenile idiopathic arthritis. RMD Open 9, e002941 (2023).
Google Scholar
Friščić, J. et al. The complement system drives local inflammatory tissue priming by metabolic reprogramming of synovial fibroblasts. Immunity 54, 1002–1021.e10 (2021).
Google Scholar
Coste, F. & Forestier, J. Hémiplégie et nodosités d’Heberden contralatèrales. Bull. Mém. Société Médicale Hôp. 51, 772 (1935).